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Tomato leaf curl Yunnan virus (TLCYnV) C4 protein interacts with BKI1, stabilizes it at the plasma membrane, and impairs ER autophosphorylation through suppressing the dissociation of the BKI1/ER complex, and then inhibits the activation of downstream MAPK cascades, which ultimately creates a favorable environment for TLCYnV infection..
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C4(R76P), C4(Q77P), C4(L78P), C4(M79P), C4(M80P), C4 (L81P), C4(L82P), C4(Q83P) and C4(R84P) were generated by overlapping PCR methods as described by Tao et al. (2002). All primers used for plasmid construction are listed in Supporting Information TableS1. Generation of viral infectious clones The generation of TLCYnV and TLCYnV mutant viral.
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Here we report that tomato leaf curl Yunnan virus (TLCYnV) C4 interacts with a glycogen synthase kinase 3 (GSK3)/SHAGGY-like kinase, designed NbSKη, in Nicotiana benthamiana. Pro32, Asn34 and Thr35 of TLCYnV C4 are critical for its interaction with NbSKη and required for C4-induced typical symptoms. Interestingly, TLCYnV C4 directs NbSKη to.
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The tomato leaf curl Yunnan virus (TLCYnV) C4 protein can impair the HIR1-mediated cell death through promoting the degradation of HIR1 via physical interaction with this host protein (Mei et al., 2019). A clear and dynamic description of the subcellular localization of viral proteins is important to understand the virus infection cycle.
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The C4 protein encoded by tomato leaf curl Yunnan virus reverses transcriptional gene silencing by interacting with NbDRM2 and impairing its DNA-binding ability. November 2020;
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Tomato leaf curl Yunnan virus (TLCYnV) C4 protein interacts with BKI1, stabilizes it at the plasma membrane and impairs ER autophosphorylation through suppressing the dissociation of the BKI1/ER complex, and then inhibits the activation of downstream MAPK cascades, which ultimately creates a favorable environment for TLCYnV infection..
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Author summary Induction of cell division is essential for DNA virus replication in differentiated plant cells. To achieve efficient replication, DNA viruses encode C4 protein to induce cell division. However, the mechanism of C4-induced cell division is still largely unknown. This study demonstrates that tomato leaf curl Yunnan virus (TLCYnV) C4 protein hijacks NbSKη, directs NbSKη to the.
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Compared with those in semi-arid grasslands and arid deserts in North China, more C4 grasses and much less Chenopodiaceae C4 species occurred in the tropical region, indicating the physiological responses of C4 plants from the two families are very different. Floristic composition, morphological functional types, and altitudinal distribution pattern for C4 species were studied in Yunnan.
Yunnanochoerus gandakasensis casts of lower molars from Yuanmou,... Download Scientific Diagram
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Yunnan, (UK: / j uː ˈ n æ n /, US: / ˌ j uː ˈ n ɑː n /; Chinese: 云南; pinyin: Yúnnán; Mandarin pronunciation: ⓘ) is a landlocked province in Southwest China.The province spans approximately 394,000 km 2 (152,000 sq mi) and has a population of 48.3 million (as of 2018). The capital of the province is Kunming.The province borders the Chinese provinces of Guizhou, Sichuan.
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The C4 protein encoded by Tomato leaf curl Yunnan virus interferes with MAPK cascade‐related defense responses through inhibiting the dissociation of the ERECTA/BKI1 complex April 2021 New.
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Here we report that tomato leaf curl Yunnan virus (TLCYnV) C4 interacts with a glycogen synthase kinase 3 (GSK3)/SHAGGY-like kinase, designed NbSKη, in Nicotiana benthamiana. Pro32, Asn34 and.